Title : Protease-activated receptor 2 deficiency mediates cardiac fibrosis and diastolic dysfunction.

Pub. Date : 2019 Oct 21

PMID : 31004144






1 Functional Relationships(s)
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1 The treatment with the PAR1 antagonist, vorapaxar, reduced cardiac fibrosis by 44% (P = 0.03) and reduced inflammation in a metabolic disease model (apolipoprotein E-ko mice). vorapaxar coagulation factor II (thrombin) receptor Mus musculus