Title : The Protection of Midazolam Against Immune Mediated Liver Injury Induced by Lipopolysaccharide and Galactosamine in Mice.

Pub. Date : 2018

PMID : 30670973






2 Functional Relationships(s)
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1 Mechanistically, midazolam suppressed tumor necrosis factor-alpha (TNF-alpha) and interleukin-1beta (IL-1beta) produced by LPS stimulated liver macrophages in vivo and bone marrow monocytes in vitro, and reduced the expression of major histocompatibility complex class II (MHC II), cluster of differentiation 40 and 86 (CD40 and CD86) on the cell surface. Midazolam tumor necrosis factor Mus musculus
2 Mechanistically, midazolam suppressed tumor necrosis factor-alpha (TNF-alpha) and interleukin-1beta (IL-1beta) produced by LPS stimulated liver macrophages in vivo and bone marrow monocytes in vitro, and reduced the expression of major histocompatibility complex class II (MHC II), cluster of differentiation 40 and 86 (CD40 and CD86) on the cell surface. Midazolam tumor necrosis factor Mus musculus