Title : Phosphorylation of human TRM9L integrates multiple stress-signaling pathways for tumor growth suppression.

Pub. Date : 2018 Jul

PMID : 30009260






1 Functional Relationships(s)
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1 Using a chemical genetic approach, we identified a key serine residue in TRM9L that undergoes hyperphosphorylation downstream of the oxidative stress-activated MEK (mitogen-activated protein kinase kinase)-ERK (extracellular signal-regulated kinase)-RSK (ribosomal protein S6 kinase) signaling cascade. Serine mitogen-activated protein kinase 1 Homo sapiens