Title : Aberrant Glycosylation Augments the Immuno-Stimulatory Activities of Soluble Calreticulin.

Pub. Date : 2018 Feb 27

PMID : 29495436






1 Functional Relationships(s)
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1 In the present study, tunicamycin, an N-glycosyltransferase inhibitor, was employed to treat CHO cells (CHO-CRT) stably expressing full-length recombinant mouse CRT in secreted form for preparation of aberrantly glycosylated eCRT (tun-eCRT). Tunicamycin calreticulin Homo sapiens