Title : Potassium intake modulates the thiazide-sensitive sodium-chloride cotransporter (NCC) activity via the Kir4.1 potassium channel.

Pub. Date : 2018 Apr

PMID : 29310825






1 Functional Relationships(s)
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1 Disruption of Kir4.1 significantly increased basal urinary sodium excretion but it abolished the natriuretic effect of hydrochlorothiazide. Hydrochlorothiazide potassium inwardly-rectifying channel, subfamily J, member 10 Mus musculus