Title : Cross Talk Between Cellular Redox State and the Antiapoptotic Protein Bcl-2.

Pub. Date : 2018 Nov 1

PMID : 29304561






1 Functional Relationships(s)
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1 However, under states of oxidative stress, overexpression of Bcl-2 functions as a redox sink to prevent excessive buildup of reactive oxygen species, thereby inhibiting execution signals. Reactive Oxygen Species BCL2 apoptosis regulator Homo sapiens