Title : Mutant FGFR3 associated with SADDAN disease causes cytoskeleton disorganization through PLCĪ³1/Src-mediated paxillin hyperphosphorylation.

Pub. Date : 2018 Feb

PMID : 29242050






2 Functional Relationships(s)
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Protein Name
Organism
1 Paxillin phosphorylation was upregulated at tyrosine 118, a functional target of Src and FAK kinases. Tyrosine protein tyrosine kinase 2 Homo sapiens
2 Moreover, we found that SADDAN-FGFR3 induced FAK phosphorylation at tyrosines 576/577, suggesting its involvement as a Src co-activator in paxillin phosphorylation. Tyrosine protein tyrosine kinase 2 Homo sapiens