Title : Tunicamycin-induced ER stress in breast cancer cells neither expresses GRP78 on the surface nor secretes it into the media.

Pub. Date : 2018 Feb 1

PMID : 29206917






1 Functional Relationships(s)
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1 Paraffin sections from N-glycosylation inhibitor tunicamycin treated ER-/PR-/HER2+ (double negative) breast tumor in athymic nude mice exhibited reduced N-glycan but increased GRP78 expression. Paraffin erb-b2 receptor tyrosine kinase 2 Homo sapiens