Title : Glucose deprivation induces primary cilium formation through mTORC1 inactivation.

Pub. Date : 2018 Jan 8

PMID : 29180513






5 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 In contrast, rapamycin treatment increased the level of the p27KIP1 (also known as CDKN1B) cyclin-dependent kinase inhibitor, and rapamycin-induced ciliogenesis was abrogated in p27KIP1-depleted cells. Sirolimus cyclin dependent kinase inhibitor 1B Homo sapiens
2 In contrast, rapamycin treatment increased the level of the p27KIP1 (also known as CDKN1B) cyclin-dependent kinase inhibitor, and rapamycin-induced ciliogenesis was abrogated in p27KIP1-depleted cells. Sirolimus cyclin dependent kinase inhibitor 1B Homo sapiens
3 In contrast, rapamycin treatment increased the level of the p27KIP1 (also known as CDKN1B) cyclin-dependent kinase inhibitor, and rapamycin-induced ciliogenesis was abrogated in p27KIP1-depleted cells. Sirolimus cyclin dependent kinase inhibitor 1B Homo sapiens
4 In contrast, rapamycin treatment increased the level of the p27KIP1 (also known as CDKN1B) cyclin-dependent kinase inhibitor, and rapamycin-induced ciliogenesis was abrogated in p27KIP1-depleted cells. Sirolimus cyclin dependent kinase inhibitor 1B Homo sapiens
5 In contrast, rapamycin treatment increased the level of the p27KIP1 (also known as CDKN1B) cyclin-dependent kinase inhibitor, and rapamycin-induced ciliogenesis was abrogated in p27KIP1-depleted cells. Sirolimus cyclin dependent kinase inhibitor 1B Homo sapiens