Title : Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.

Pub. Date : 2018 Jan 5

PMID : 29127203






1 Functional Relationships(s)
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1 We examined these samples for systemic and brain inflammation; amyloid-beta peptide (Abeta) and Ser-202-phosphorylated Tau (p-TauSer-202) levels; and RAGE, RAGE ligands, and RAGE intracellular signaling. Serine microtubule associated protein tau Homo sapiens