Title : Tonabersat Prevents Inflammatory Damage in the Central Nervous System by Blocking Connexin43 Hemichannels.

Pub. Date : 2017 Oct

PMID : 28560708






4 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Both connexin43 hemichannels and pannexin channels released adenosine triphosphate (ATP) during ischemia in an in vitro ischemia model, but only connexin43 hemichannels contributed to ATP release during reperfusion. Adenosine Triphosphate gap junction protein alpha 1 Homo sapiens
2 Both connexin43 hemichannels and pannexin channels released adenosine triphosphate (ATP) during ischemia in an in vitro ischemia model, but only connexin43 hemichannels contributed to ATP release during reperfusion. Adenosine Triphosphate gap junction protein alpha 1 Homo sapiens
3 Both connexin43 hemichannels and pannexin channels released adenosine triphosphate (ATP) during ischemia in an in vitro ischemia model, but only connexin43 hemichannels contributed to ATP release during reperfusion. Adenosine Triphosphate gap junction protein alpha 1 Homo sapiens
4 Tonabersat inhibited connexin43 hemichannel-mediated ATP release during both ischemia and reperfusion phases, with direct channel block confirmed using electrophysiology. Adenosine Triphosphate gap junction protein alpha 1 Homo sapiens