Title : Activation of PERK Elicits Memory Impairment through Inactivation of CREB and Downregulation of PSD95 After Traumatic Brain Injury.

Pub. Date : 2017 Jun 14

PMID : 28522733






1 Functional Relationships(s)
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1 Similarly, administration of either GSK2656157 or overexpression of PERK-K618A in primary neurons rescues the loss of dendritic outgrowth and number of synapses after treatment with a PERK activator, tunicamycin. Tunicamycin eukaryotic translation initiation factor 2 alpha kinase 3 Homo sapiens