Title : Fatty acid amide hydrolase (FAAH) regulates hypercapnia/ischemia-induced increases in n-acylethanolamines in mouse brain.

Pub. Date : 2017 Sep

PMID : 28488728






4 Functional Relationships(s)
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1 N-acylethanolamines (NAEs) are endogenous lipid ligands for several receptors including cannabinoid receptors and peroxisome proliferator-activated receptor-alpha (PPAR-alpha), which regulate numerous physiological functions. N-acylethanolamines peroxisome proliferator activated receptor alpha Mus musculus
2 N-acylethanolamines (NAEs) are endogenous lipid ligands for several receptors including cannabinoid receptors and peroxisome proliferator-activated receptor-alpha (PPAR-alpha), which regulate numerous physiological functions. N-acylethanolamines peroxisome proliferator activated receptor alpha Mus musculus
3 N-acylethanolamines (NAEs) are endogenous lipid ligands for several receptors including cannabinoid receptors and peroxisome proliferator-activated receptor-alpha (PPAR-alpha), which regulate numerous physiological functions. N-acylethanolamines peroxisome proliferator activated receptor alpha Mus musculus
4 N-acylethanolamines (NAEs) are endogenous lipid ligands for several receptors including cannabinoid receptors and peroxisome proliferator-activated receptor-alpha (PPAR-alpha), which regulate numerous physiological functions. N-acylethanolamines peroxisome proliferator activated receptor alpha Mus musculus