Title : The RB-IL-6 axis controls self-renewal and endocrine therapy resistance by fine-tuning mitochondrial activity.

Pub. Date : 2017 Sep 7

PMID : 28481867






1 Functional Relationships(s)
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1 The mechanism whereby RB inactivation increases IL-6 production in MCF-7 cells appeared to involve fatty acid oxidation (FAO)-dependent mitochondrial metabolism and c-Jun NH(2)-terminal kinase (JNK). Fatty Acids interleukin 6 Homo sapiens