Title : Synergistic killing of FLT3ITD-positive AML cells by combined inhibition of tyrosine-kinase activity and N-glycosylation.

Pub. Date : 2017 Apr 18

PMID : 28460451






2 Functional Relationships(s)
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1 In addition, tunicamycin caused pronounced endoplasmatic reticulum stress and apoptosis through activation of protein kinase RNA-like endoplasmic reticulum kinase (PERK) and activation of the gene encoding CCAAT-enhancer-binding protein homologous protein (CHOP). Tunicamycin eukaryotic translation initiation factor 2 alpha kinase 3 Homo sapiens
2 In addition, tunicamycin caused pronounced endoplasmatic reticulum stress and apoptosis through activation of protein kinase RNA-like endoplasmic reticulum kinase (PERK) and activation of the gene encoding CCAAT-enhancer-binding protein homologous protein (CHOP). Tunicamycin eukaryotic translation initiation factor 2 alpha kinase 3 Homo sapiens