Title : Impairment of endoplasmic reticulum is involved in β-cell dysfunction induced by microcystin-LR.

Pub. Date : 2017 Apr

PMID : 28162804






4 Functional Relationships(s)
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1 Furthermore, MC-LR-induced ER stress significantly promoted the expression of PERK/eIF2alpha and their downstream targets (ATF4, CHOP, and Gadd34), which indicates that PERK-eIF2alpha-ATF4 pathway is involved in MC-LR-induced insulin deficiency. cyanoginosin LR activating transcription factor 4 Homo sapiens
2 Furthermore, MC-LR-induced ER stress significantly promoted the expression of PERK/eIF2alpha and their downstream targets (ATF4, CHOP, and Gadd34), which indicates that PERK-eIF2alpha-ATF4 pathway is involved in MC-LR-induced insulin deficiency. cyanoginosin LR activating transcription factor 4 Homo sapiens
3 Furthermore, MC-LR-induced ER stress significantly promoted the expression of PERK/eIF2alpha and their downstream targets (ATF4, CHOP, and Gadd34), which indicates that PERK-eIF2alpha-ATF4 pathway is involved in MC-LR-induced insulin deficiency. cyanoginosin LR activating transcription factor 4 Homo sapiens
4 Furthermore, MC-LR-induced ER stress significantly promoted the expression of PERK/eIF2alpha and their downstream targets (ATF4, CHOP, and Gadd34), which indicates that PERK-eIF2alpha-ATF4 pathway is involved in MC-LR-induced insulin deficiency. cyanoginosin LR activating transcription factor 4 Homo sapiens