Pub. Date : 2016 Nov
PMID : 27666407
6 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | Depression accelerates the development of gastric cancer through reactive oxygen species-activated ABL1 (Review). | Reactive Oxygen Species | ABL proto-oncogene 1, non-receptor tyrosine kinase | Homo sapiens |
| 2 | Recent evidence indicates that ABL1 was dysregulated in both major depressive disorder (MDD) and cancer patients with depression, and high levels of reactive oxygen species (ROS) can lead to the activation of ABL1 in response to OS and that activated ABL1 subsequently contributes to development of GC via interactions with the downstream targets and corresponding signaling pathways. | Reactive Oxygen Species | ABL proto-oncogene 1, non-receptor tyrosine kinase | Homo sapiens |
| 3 | Recent evidence indicates that ABL1 was dysregulated in both major depressive disorder (MDD) and cancer patients with depression, and high levels of reactive oxygen species (ROS) can lead to the activation of ABL1 in response to OS and that activated ABL1 subsequently contributes to development of GC via interactions with the downstream targets and corresponding signaling pathways. | Reactive Oxygen Species | ABL proto-oncogene 1, non-receptor tyrosine kinase | Homo sapiens |
| 4 | Recent evidence indicates that ABL1 was dysregulated in both major depressive disorder (MDD) and cancer patients with depression, and high levels of reactive oxygen species (ROS) can lead to the activation of ABL1 in response to OS and that activated ABL1 subsequently contributes to development of GC via interactions with the downstream targets and corresponding signaling pathways. | Reactive Oxygen Species | ABL proto-oncogene 1, non-receptor tyrosine kinase | Homo sapiens |
| 5 | Recent evidence indicates that ABL1 was dysregulated in both major depressive disorder (MDD) and cancer patients with depression, and high levels of reactive oxygen species (ROS) can lead to the activation of ABL1 in response to OS and that activated ABL1 subsequently contributes to development of GC via interactions with the downstream targets and corresponding signaling pathways. | Reactive Oxygen Species | ABL proto-oncogene 1, non-receptor tyrosine kinase | Homo sapiens |
| 6 | Recent evidence indicates that ABL1 was dysregulated in both major depressive disorder (MDD) and cancer patients with depression, and high levels of reactive oxygen species (ROS) can lead to the activation of ABL1 in response to OS and that activated ABL1 subsequently contributes to development of GC via interactions with the downstream targets and corresponding signaling pathways. | Reactive Oxygen Species | ABL proto-oncogene 1, non-receptor tyrosine kinase | Homo sapiens |