Title : Sulfur Dioxide Inhibits Extracellular Signal-regulated Kinase Signaling to Attenuate Vascular Smooth Muscle Cell Proliferation in Angiotensin II-induced Hypertensive Mice.

Pub. Date : 2016 Sep 20

PMID : 27625096






4 Functional Relationships(s)
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1 Sulfur Dioxide Inhibits Extracellular Signal-regulated Kinase Signaling to Attenuate Vascular Smooth Muscle Cell Proliferation in Angiotensin II-induced Hypertensive Mice. Sulfur Dioxide angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
2 Previous research has shown that the gaseous signaling molecule sulfur dioxide (SO2) inhibits VSMC proliferation, but the mechanism for the inhibition of the angiotensin II (AngII)-induced VSMC proliferation by SO2has not been fully elucidated. Sulfur Dioxide angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
3 Previous research has shown that the gaseous signaling molecule sulfur dioxide (SO2) inhibits VSMC proliferation, but the mechanism for the inhibition of the angiotensin II (AngII)-induced VSMC proliferation by SO2has not been fully elucidated. Sulfur Dioxide angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
4 SO2, however, reduced the systemic hypertension and the wall thickness induced by AngII (P < 0.05). Sulfur Dioxide angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus