Title : Endoplasmic reticulum stress induced by tunicamycin increases resistin messenger ribonucleic acid through the pancreatic endoplasmic reticulum eukaryotic initiation factor 2α kinase-activating transcription factor 4-CAAT/enhancer binding protein-α homologous protein pathway in THP-1 human monocytes.

Pub. Date : 2016 May

PMID : 27330716






2 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 The knockdown of either PERK, activating transcription factor 4 (ATF4) or CHOP reduced tunicamycin-induced resistin mRNA. Tunicamycin DNA damage inducible transcript 3 Homo sapiens
2 CONCLUSIONS: Endoplasmic reticulum stress induced by tunicamycin increased resistin mRNA through the PERK-ATF4-CHOP pathway in THP-1 human monocytes. Tunicamycin DNA damage inducible transcript 3 Homo sapiens