Title : Inhibition of endoplasmic reticulum (ER) stress sensors sensitizes cancer stem-like cells to ER stress-mediated apoptosis.

Pub. Date : 2016 Aug 9

PMID : 27322083






1 Functional Relationships(s)
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1 Biochemical assays demonstrated that sphere-forming cells were shifted to pro-survival signaling through the inactivation of IRE1 (XBP-1 splicing) and activation of PERK (elF2alpha phosphorylation) branches under tunicamycin-induced ER stress conditions. Tunicamycin endoplasmic reticulum to nucleus signaling 1 Homo sapiens