Title : Melatonin protects podocytes from angiotensin II-induced injury in an in vitro diabetic nephropathy model.

Pub. Date : 2016 Jul

PMID : 27220903






2 Functional Relationships(s)
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1 Based on an in vitro podocyte injury model, the present study found that the application of melatonin significantly reduced AngII-induced apoptosis and increased the proliferative rate of cells, as evidenced by decreased expression levels of apoptotic proteins, including Caspase-3 and Bax, and a change in the ratio of Bax/Bcl-2. Melatonin BCL2 associated X, apoptosis regulator Homo sapiens
2 Based on an in vitro podocyte injury model, the present study found that the application of melatonin significantly reduced AngII-induced apoptosis and increased the proliferative rate of cells, as evidenced by decreased expression levels of apoptotic proteins, including Caspase-3 and Bax, and a change in the ratio of Bax/Bcl-2. Melatonin BCL2 associated X, apoptosis regulator Homo sapiens