Title : Particulate Matter Facilitates C6 Glioma Cells Activation and the Release of Inflammatory Factors Through MAPK and JAK2/STAT3 Pathways.

Pub. Date : 2016 Aug

PMID : 27068033






3 Functional Relationships(s)
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1 In addition, it was demonstrated that inhibition of p38, JNK and JAK prevented STAT3 tyrosine phosphorylation induced by PM, while blocking ERK did not. Tyrosine mitogen-activated protein kinase 8 Rattus norvegicus
2 MAPKs (p38 and JNK) could regulate tyrosine STAT3 phosphorylation, which suggested that the JAK2/STAT3 pathway might be the downstream of p38/JNK MAPK pathways. Tyrosine mitogen-activated protein kinase 8 Rattus norvegicus
3 MAPKs (p38 and JNK) could regulate tyrosine STAT3 phosphorylation, which suggested that the JAK2/STAT3 pathway might be the downstream of p38/JNK MAPK pathways. Tyrosine mitogen-activated protein kinase 8 Rattus norvegicus