Title : Ptp1b deletion in pro-opiomelanocortin neurons increases energy expenditure and impairs endothelial function via TNF-α dependent mechanisms.

Pub. Date : 2016 Jun 1

PMID : 26935109






1 Functional Relationships(s)
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1 Nitric oxide synthase inhibition [N-nitro-L-arginine methyl ester (L-NAME)] reduced relaxation to a similar extent in Ptp1b+/+ and POMC-Ptp1b-/- mice. NG-Nitroarginine Methyl Ester pro-opiomelanocortin-alpha Mus musculus