Title : Ecto-5'-nucleotidase CD73 modulates the innate immune response to influenza infection but is not required for development of influenza-induced acute lung injury.

Pub. Date : 2015 Dec 1

PMID : 26432867






4 Functional Relationships(s)
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1 Because AMP hydrolysis by the ecto-5"-nucleotidase (CD73) plays a central role in and is rate-limiting for generation of adenosine in the normal lung, we hypothesized that ALI would be attenuated in C57BL/6-congenic CD73-knockout (CD73-KO) mice. Adenosine Monophosphate 5' nucleotidase, ecto Mus musculus
2 Because AMP hydrolysis by the ecto-5"-nucleotidase (CD73) plays a central role in and is rate-limiting for generation of adenosine in the normal lung, we hypothesized that ALI would be attenuated in C57BL/6-congenic CD73-knockout (CD73-KO) mice. Adenosine Monophosphate 5' nucleotidase, ecto Mus musculus
3 Because AMP hydrolysis by the ecto-5"-nucleotidase (CD73) plays a central role in and is rate-limiting for generation of adenosine in the normal lung, we hypothesized that ALI would be attenuated in C57BL/6-congenic CD73-knockout (CD73-KO) mice. Adenosine Monophosphate 5' nucleotidase, ecto Mus musculus
4 Because AMP hydrolysis by the ecto-5"-nucleotidase (CD73) plays a central role in and is rate-limiting for generation of adenosine in the normal lung, we hypothesized that ALI would be attenuated in C57BL/6-congenic CD73-knockout (CD73-KO) mice. Adenosine Monophosphate 5' nucleotidase, ecto Mus musculus