Title : Selective Targeting of CTNBB1-, KRAS- or MYC-Driven Cell Growth by Combinations of Existing Drugs.

Pub. Date : 2015

PMID : 26018524






1 Functional Relationships(s)
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1 The EGFR/ERBB2 inhibitor neratinib acted synergistically with the spindle poison docetaxel and with the Aurora kinase inhibitor GSK-1070916 in cell lines with MYC amplification. Docetaxel epidermal growth factor receptor Homo sapiens