Title : Hypertension enhances Aβ-induced neurovascular dysfunction, promotes β-secretase activity, and leads to amyloidogenic processing of APP.

Pub. Date : 2016 Jan

PMID : 25920959






5 Functional Relationships(s)
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1 Infusion of angiotensin II (ANGII; intravenously) elevated blood pressure and attenuated the cerebral blood flow (CBF) response to whisker stimulation or the endothelium-dependent vasodilator acetylcholine (ACh) (P < 0.05). Acetylcholine angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
2 Infusion of angiotensin II (ANGII; intravenously) elevated blood pressure and attenuated the cerebral blood flow (CBF) response to whisker stimulation or the endothelium-dependent vasodilator acetylcholine (ACh) (P < 0.05). Acetylcholine angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
3 Infusion of angiotensin II (ANGII; intravenously) elevated blood pressure and attenuated the cerebral blood flow (CBF) response to whisker stimulation or the endothelium-dependent vasodilator acetylcholine (ACh) (P < 0.05). Acetylcholine angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
4 Infusion of angiotensin II (ANGII; intravenously) elevated blood pressure and attenuated the cerebral blood flow (CBF) response to whisker stimulation or the endothelium-dependent vasodilator acetylcholine (ACh) (P < 0.05). Acetylcholine angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
5 Neocortical application of Abeta in mice receiving ANGII worsened the responses to ACh (P < 0.05). Acetylcholine angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus