Title : Anticancer effect of celastrol on human triple negative breast cancer: possible involvement of oxidative stress, mitochondrial dysfunction, apoptosis and PI3K/Akt pathways.

Pub. Date : 2015 Jun

PMID : 25818165






1 Functional Relationships(s)
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1 Meanwhile, celastrol triggered reactive oxygen species production with collapse of mitochondrial membrane potential, down-regulation of Bcl-2 and up-regulation of Bax expression. celastrol BCL2 apoptosis regulator Homo sapiens