Title : Acetylcholine inhibits tumor necrosis factor α activated endoplasmic reticulum apoptotic pathway via EGFR-PI3K signaling in cardiomyocytes.

Pub. Date : 2015 Apr

PMID : 25201632






8 Functional Relationships(s)
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1 Acetylcholine inhibits tumor necrosis factor alpha activated endoplasmic reticulum apoptotic pathway via EGFR-PI3K signaling in cardiomyocytes. Acetylcholine tumor necrosis factor Rattus norvegicus
2 Previous findings have shown that acetylcholine (ACh) decreased hypoxia-induced tumor necrosis factor alpha (TNF alpha) production, thus protected against cardiomyocyte injury. Acetylcholine tumor necrosis factor Rattus norvegicus
3 Previous findings have shown that acetylcholine (ACh) decreased hypoxia-induced tumor necrosis factor alpha (TNF alpha) production, thus protected against cardiomyocyte injury. Acetylcholine tumor necrosis factor Rattus norvegicus
4 Previous findings have shown that acetylcholine (ACh) decreased hypoxia-induced tumor necrosis factor alpha (TNF alpha) production, thus protected against cardiomyocyte injury. Acetylcholine tumor necrosis factor Rattus norvegicus
5 Previous findings have shown that acetylcholine (ACh) decreased hypoxia-induced tumor necrosis factor alpha (TNF alpha) production, thus protected against cardiomyocyte injury. Acetylcholine tumor necrosis factor Rattus norvegicus
6 However, whether and how ACh affects TNF alpha-induced endoplasmic reticulum (ER) stress and cell apoptosis remain poorly defined. Acetylcholine tumor necrosis factor Rattus norvegicus
7 This study was aimed at determining the effect of ACh in H9c2 cells after TNF alpha stimulation. Acetylcholine tumor necrosis factor Rattus norvegicus
8 Exogenously administered ACh significantly decreased these TNF alpha-induced changes. Acetylcholine tumor necrosis factor Rattus norvegicus