Title : Nicotine mediates oxidative stress and apoptosis through cross talk between NOX1 and Bcl-2 in lung epithelial cells.

Pub. Date : 2014 Nov

PMID : 25151121






5 Functional Relationships(s)
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1 Nicotine mediates oxidative stress and apoptosis through cross talk between NOX1 and Bcl-2 in lung epithelial cells. Nicotine BCL2 apoptosis regulator Homo sapiens
2 Overexpression of Bcl-2 completely prevented nicotine"s detrimental effects, suggesting Bcl-2as a downstream key regulator in nicotine/NOX1-induced cell damage. Nicotine BCL2 apoptosis regulator Homo sapiens
3 Overexpression of Bcl-2 completely prevented nicotine"s detrimental effects, suggesting Bcl-2as a downstream key regulator in nicotine/NOX1-induced cell damage. Nicotine BCL2 apoptosis regulator Homo sapiens
4 Overexpression of Bcl-2 completely prevented nicotine"s detrimental effects, suggesting Bcl-2as a downstream key regulator in nicotine/NOX1-induced cell damage. Nicotine BCL2 apoptosis regulator Homo sapiens
5 Overexpression of Bcl-2 completely prevented nicotine"s detrimental effects, suggesting Bcl-2as a downstream key regulator in nicotine/NOX1-induced cell damage. Nicotine BCL2 apoptosis regulator Homo sapiens