Title : Activation of the EGFR/p38/JNK pathway by mitochondrial-derived hydrogen peroxide contributes to oxygen-induced contraction of ductus arteriosus.

Pub. Date : 2014 Sep

PMID : 24906456






3 Functional Relationships(s)
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1 Anisomycin, an activator of EGFR"s downstream kinases, p38 and JNK, caused DA contraction; conversely, oxygen-induced DA contraction was blocked by inhibitors of p38 mitogen-activated protein kinases (MAPK) (SB203580) or JNK (JNK inhibitor II). Anisomycin mitogen-activated protein kinase 8 Homo sapiens
2 Anisomycin, an activator of EGFR"s downstream kinases, p38 and JNK, caused DA contraction; conversely, oxygen-induced DA contraction was blocked by inhibitors of p38 mitogen-activated protein kinases (MAPK) (SB203580) or JNK (JNK inhibitor II). Anisomycin mitogen-activated protein kinase 8 Homo sapiens
3 Anisomycin, an activator of EGFR"s downstream kinases, p38 and JNK, caused DA contraction; conversely, oxygen-induced DA contraction was blocked by inhibitors of p38 mitogen-activated protein kinases (MAPK) (SB203580) or JNK (JNK inhibitor II). Anisomycin mitogen-activated protein kinase 8 Homo sapiens