Title : Tumor necrosis factor/cachectin plays a key role in bleomycin-induced pneumopathy and fibrosis.

Pub. Date : 1989 Sep 1

PMID : 2475571






9 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Tumor necrosis factor/cachectin plays a key role in bleomycin-induced pneumopathy and fibrosis. Bleomycin tumor necrosis factor Mus musculus
2 Tumor necrosis factor/cachectin plays a key role in bleomycin-induced pneumopathy and fibrosis. Bleomycin tumor necrosis factor Mus musculus
3 The role of TNF-alpha/cachectin in the pneumopathy elicited by bleomycin has been investigated. Bleomycin tumor necrosis factor Mus musculus
4 The role of TNF-alpha/cachectin in the pneumopathy elicited by bleomycin has been investigated. Bleomycin tumor necrosis factor Mus musculus
5 After a single intratracheal bleomycin instillation, an increase of the lung TNF-alpha mRNA level was evident, from days 5 to 15, as shown by Northern gel analysis of whole lung RNA. Bleomycin tumor necrosis factor Mus musculus
6 In mice passively immunized with rabbit anti-mouse TNF-alpha IgG, the bleomycin-induced collagen deposition, evaluated by the total lung hydroxyproline assay on day 15, was prevented. Bleomycin tumor necrosis factor Mus musculus
7 Depletion of the CD4 and CD8 T lymphocytes by an in vivo treatment with mAb prevented the bleomycin-induced increase of TNF mRNA level and fibrosis. Bleomycin tumor necrosis factor Mus musculus
8 After an administration of bleomycin in continuous intraperitoneal perfusion, the diffuse alveolar damage observed by light and electron microscopy was almost completely prevented by anti-TNF antibody. Bleomycin tumor necrosis factor Mus musculus
9 These results indicate that in response to bleomycin, the T lymphocytes induce, by an undefined mechanism, an increase of the pulmonary TNF production, which leads to alveolar damage, growth of fibroblast, and collagen deposition. Bleomycin tumor necrosis factor Mus musculus