Title : (-)-Epigallocatechin-3-gallate ameliorates photodynamic therapy responses in an in vitro T lymphocyte model.

Pub. Date : 2014 Oct

PMID : 24700514






3 Functional Relationships(s)
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1 The EGCG also enhanced intracellular reactive oxygen species accumulation, c-Jun N-terminal kinase (JNK) phosphorylation, and interferon-gamma (IFN-gamma) gene expression, all of which are involved in PhB-induced apoptosis. epigallocatechin gallate interferon gamma Homo sapiens
2 The EGCG also enhanced intracellular reactive oxygen species accumulation, c-Jun N-terminal kinase (JNK) phosphorylation, and interferon-gamma (IFN-gamma) gene expression, all of which are involved in PhB-induced apoptosis. epigallocatechin gallate interferon gamma Homo sapiens
3 Taken together, our data suggest that EGCG is capable of potentiating photodynamic therapy responses, presumably through the intracellular oxidative stress-sensitive JNK/IFN-gamma pathway by exogenous hydrogen peroxide formation. epigallocatechin gallate interferon gamma Homo sapiens