Title : Ah receptor-mediated suppression of liver regeneration through NC-XRE-driven p21Cip1 expression.

Pub. Date : 2014 Apr

PMID : 24431146






3 Functional Relationships(s)
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1 Previous studies in hepatocyte-derived cell lines and the whole liver established that the aryl hydrocarbon receptor (AhR) can disrupt G1-phase cell cycle progression following exposure to persistent AhR agonists, such as TCDD (dioxin, 2,3,7,8-tetrachlorodibenzo-p-dioxin). Dioxins aryl-hydrocarbon receptor Mus musculus
2 Previous studies in hepatocyte-derived cell lines and the whole liver established that the aryl hydrocarbon receptor (AhR) can disrupt G1-phase cell cycle progression following exposure to persistent AhR agonists, such as TCDD (dioxin, 2,3,7,8-tetrachlorodibenzo-p-dioxin). Dioxins aryl-hydrocarbon receptor Mus musculus
3 Previous studies in hepatocyte-derived cell lines and the whole liver established that the aryl hydrocarbon receptor (AhR) can disrupt G1-phase cell cycle progression following exposure to persistent AhR agonists, such as TCDD (dioxin, 2,3,7,8-tetrachlorodibenzo-p-dioxin). Dioxins aryl-hydrocarbon receptor Mus musculus