Title : Tyrosine kinase inhibition increases functional parkin-Beclin-1 interaction and enhances amyloid clearance and cognitive performance.

Pub. Date : 2013 Aug

PMID : 23737459






1 Functional Relationships(s)
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1 Isolation of autophagic vacuoles (AVs) in AD mouse brain shows accumulation of parkin and amyloid, consistent with previous results in AD brains, while Bosutinib and Nilotinib increase parkin-Beclin-1 interaction and result in protein deposition in the lysosome. nilotinib beclin 1, autophagy related Mus musculus