Title : A feedback loop in PPARĪ³-adenosine A2A receptor signaling inhibits inflammation and attenuates lung damages in a mouse model of LPS-induced acute lung injury.

Pub. Date : 2013 Sep

PMID : 23712033






4 Functional Relationships(s)
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Protein Name
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1 Instead of directly interacting with PPARgamma, A2AR stimulated PPARgamma expression via protein kinase A (PKA)-cAMP response element binding protein (CREB) signaling by provoking the binding of CREB to a cAMP responsive element (CRE)-like site in PPARgamma gene promoter region. Cyclic AMP peroxisome proliferator activated receptor gamma Mus musculus
2 Instead of directly interacting with PPARgamma, A2AR stimulated PPARgamma expression via protein kinase A (PKA)-cAMP response element binding protein (CREB) signaling by provoking the binding of CREB to a cAMP responsive element (CRE)-like site in PPARgamma gene promoter region. Cyclic AMP peroxisome proliferator activated receptor gamma Mus musculus
3 Instead of directly interacting with PPARgamma, A2AR stimulated PPARgamma expression via protein kinase A (PKA)-cAMP response element binding protein (CREB) signaling by provoking the binding of CREB to a cAMP responsive element (CRE)-like site in PPARgamma gene promoter region. Cyclic AMP peroxisome proliferator activated receptor gamma Mus musculus
4 Instead of directly interacting with PPARgamma, A2AR stimulated PPARgamma expression via protein kinase A (PKA)-cAMP response element binding protein (CREB) signaling by provoking the binding of CREB to a cAMP responsive element (CRE)-like site in PPARgamma gene promoter region. Cyclic AMP peroxisome proliferator activated receptor gamma Mus musculus