Title : Cigarette smoke condensate extracts induce IL-1-beta production from rheumatoid arthritis patient-derived synoviocytes, but not osteoarthritis patient-derived synoviocytes, through aryl hydrocarbon receptor-dependent NF-kappa-B activation and novel NF-kappa-B sites.

Pub. Date : 2013 Jun

PMID : 23452206






2 Functional Relationships(s)
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1 CSC-induced NF-kappaB activation, IL-1beta promoter activity, IL-1beta mRNA upregulation, and CYP1A1 mRNA induction were all inhibited by an aryl hydrocarbon receptor (AhR) antagonist alpha-naphthoflavone. alpha-naphthoflavone aryl hydrocarbon receptor Homo sapiens
2 CSC-induced NF-kappaB activation, IL-1beta promoter activity, IL-1beta mRNA upregulation, and CYP1A1 mRNA induction were all inhibited by an aryl hydrocarbon receptor (AhR) antagonist alpha-naphthoflavone. alpha-naphthoflavone aryl hydrocarbon receptor Homo sapiens