Title : Glyceollin improves endoplasmic reticulum stress-induced insulin resistance through CaMKK-AMPK pathway in L6 myotubes.

Pub. Date : 2013 Jun

PMID : 23313043






3 Functional Relationships(s)
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1 An insulin-resistant state was induced by exposing cells to 5mug/ml of tunicamycin as indicated by decreased insulin-mediated tyrosine phosphorylation of insulin receptor substrate-1 (IRS-1) and glucose uptake. Tyrosine insulin receptor substrate 1 Mus musculus
2 An insulin-resistant state was induced by exposing cells to 5mug/ml of tunicamycin as indicated by decreased insulin-mediated tyrosine phosphorylation of insulin receptor substrate-1 (IRS-1) and glucose uptake. Tyrosine insulin receptor substrate 1 Mus musculus
3 Inhibition of insulin-mediated tyrosine phosphorylation of IRS-1 and glucose uptake under ER stress was prevented by glyceollin. Tyrosine insulin receptor substrate 1 Mus musculus