Title : Early postinjury exercise reverses memory deficits and retards the progression of closed-head injury in mice.

Pub. Date : 2013 Feb 15

PMID : 23184513






2 Functional Relationships(s)
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1 Exercise gradually restored the impact-abolished hippocampal expression of BDNF and MPK-1, while oral administration of triptolide (a synthesis inhibitor of MKP-1 and an antagonist of nuclear factor-B) before each bout of exercise blocked the restorative effects of exercise on MKP-1 and recognition memory, as well as the exercise-induced retardation of neuronal loss. triptolide dual specificity phosphatase 1 Mus musculus
2 Exercise gradually restored the impact-abolished hippocampal expression of BDNF and MPK-1, while oral administration of triptolide (a synthesis inhibitor of MKP-1 and an antagonist of nuclear factor-B) before each bout of exercise blocked the restorative effects of exercise on MKP-1 and recognition memory, as well as the exercise-induced retardation of neuronal loss. triptolide dual specificity phosphatase 1 Mus musculus