Title : Anisomycin induces apoptosis of glucocorticoid resistant acute lymphoblastic leukemia CEM-C1 cells via activation of mitogen-activated protein kinases p38 and JNK.

Pub. Date : 2013

PMID : 23067223






4 Functional Relationships(s)
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1 Anisomycin induces apoptosis of glucocorticoid resistant acute lymphoblastic leukemia CEM-C1 cells via activation of mitogen-activated protein kinases p38 and JNK. Anisomycin mitogen-activated protein kinase 8 Homo sapiens
2 The rapid up-regulation of phosphorylated mitogen-activated protein kinases (MAPKs) p38 and Jun N-terminal kinase (JNK) were observed after CEM-C1 cells were incubated with anisomycin. Anisomycin mitogen-activated protein kinase 8 Homo sapiens
3 The rapid up-regulation of phosphorylated mitogen-activated protein kinases (MAPKs) p38 and Jun N-terminal kinase (JNK) were observed after CEM-C1 cells were incubated with anisomycin. Anisomycin mitogen-activated protein kinase 8 Homo sapiens
4 These results suggested that anisomycin induced apoptosis of CEM-C1 cells via activation of p38 and JNK, and might be an attractive new agent for treatment of GC-resistant ALL. Anisomycin mitogen-activated protein kinase 8 Homo sapiens