Title : Hereditary conjugated hyperbilirubinaemia: 37 years later.

Pub. Date : 2013 Feb

PMID : 22982575






2 Functional Relationships(s)
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1 Thus, disruption of hepatic reuptake of bilirubin glucuronide due to coexisting OATP1B1 and OATP1B3 deficiencies explains Rotor-type hyperbilirubinemia.Moreover, OATP1B1 and OATP1B3 null mutations may confer substantial drug toxicity risks. Bilirubin solute carrier organic anion transporter family member 1B1 Homo sapiens
2 Thus, disruption of hepatic reuptake of bilirubin glucuronide due to coexisting OATP1B1 and OATP1B3 deficiencies explains Rotor-type hyperbilirubinemia.Moreover, OATP1B1 and OATP1B3 null mutations may confer substantial drug toxicity risks. Bilirubin solute carrier organic anion transporter family member 1B1 Homo sapiens