Title : PPARγ delivered by Ch-GNPs onto titanium surfaces inhibits implant-induced inflammation and induces bone mineralization of MC-3T3E1 osteoblast-like cells.

Pub. Date : 2013 Oct

PMID : 22713176






1 Functional Relationships(s)
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1 The inhibition mechanism of Ch-GNPs/PPARgamma was due to inhibition of both reactive oxygen species (ROS) and nitric oxide (NO) secretion (n = 3; P < 0.05). Nitric Oxide peroxisome proliferator activated receptor gamma Mus musculus