Title : Complete OATP1B1 and OATP1B3 deficiency causes human Rotor syndrome by interrupting conjugated bilirubin reuptake into the liver.

Pub. Date : 2012 Feb

PMID : 22232210






1 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Thus, disruption of hepatic reuptake of bilirubin glucuronide due to coexisting OATP1B1 and OATP1B3 deficiencies explains Rotor-type hyperbilirubinemia. Glucuronides solute carrier organic anion transporter family member 1B1 Homo sapiens