Title : RIP1 mediates the protection of geldanamycin on neuronal injury induced by oxygen-glucose deprivation combined with zVAD in primary cortical neurons.

Pub. Date : 2012 Jan

PMID : 21985437






4 Functional Relationships(s)
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Protein Name
Organism
1 RIP1 mediates the protection of geldanamycin on neuronal injury induced by oxygen-glucose deprivation combined with zVAD in primary cortical neurons. benzyloxycarbonylvalyl-alanyl-aspartyl fluoromethyl ketone receptor interacting serine/threonine kinase 1 Homo sapiens
2 In this study, we showed that oxygen-glucose deprivation (OGD) combined with a caspase inhibitor zVAD (OGD/zVAD)-induced RIP1 protein expression in a time-dependent manner. benzyloxycarbonylvalyl-alanyl-aspartyl fluoromethyl ketone receptor interacting serine/threonine kinase 1 Homo sapiens
3 In this study, we showed that oxygen-glucose deprivation (OGD) combined with a caspase inhibitor zVAD (OGD/zVAD)-induced RIP1 protein expression in a time-dependent manner. benzyloxycarbonylvalyl-alanyl-aspartyl fluoromethyl ketone receptor interacting serine/threonine kinase 1 Homo sapiens
4 We concluded that the GA-mediated protection against OGD/zVAD-induced neuronal injury was associated with enhanced RIP1 protein instability by decreasing Hsp90 protein level. benzyloxycarbonylvalyl-alanyl-aspartyl fluoromethyl ketone receptor interacting serine/threonine kinase 1 Homo sapiens