Title : Hydrogen inhalation reduced epithelial apoptosis in ventilator-induced lung injury via a mechanism involving nuclear factor-kappa B activation.

Pub. Date : 2011 May 6

PMID : 21473852






6 Functional Relationships(s)
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1 Hydrogen inhalation reduced epithelial apoptosis in ventilator-induced lung injury via a mechanism involving nuclear factor-kappa B activation. Hydrogen nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus
2 Hydrogen gas inhalation increased NFkappaB DNA binding after 1h of ventilation and decreased NFkappaB DNA binding after 2h of ventilation, as compared with controls. Hydrogen nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus
3 Hydrogen gas inhalation increased NFkappaB DNA binding after 1h of ventilation and decreased NFkappaB DNA binding after 2h of ventilation, as compared with controls. Hydrogen nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus
4 Hydrogen gas inhalation increased NFkappaB DNA binding after 1h of ventilation and decreased NFkappaB DNA binding after 2h of ventilation, as compared with controls. Hydrogen nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus
5 The early activation of NFkappaB during hydrogen treatment was correlated with elevated levels of the antiapoptotic protein Bcl-2 and decreased levels of Bax. Hydrogen nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus
6 NFkappaB activation and an associated increase in the expression of Bcl-2 may contribute, in part, to the cytoprotective effects of hydrogen against apoptotic and inflammatory signaling pathway activation during VILI. Hydrogen nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus