Title : Over-expression of NYGGF4 (PID1) inhibits glucose transport in skeletal myotubes by blocking the IRS1/PI3K/AKT insulin pathway.

Pub. Date : 2011 Mar

PMID : 21185755

1 Functional Relationships(s)
Compound Name
Protein Name
1 It also diminished insulin-stimulated tyrosine phosphorylation of IRS1 and serine phosphorylation of Akt without affecting the phosphorylation of IR, ERK1/2, p38, or JNK. Tyrosine insulin receptor substrate 1 Rattus norvegicus