Title : High salt intake delayed angiotensin II-induced hypertension in mice with a genetic variant of NADPH oxidase.

Pub. Date : 2011 Jan

PMID : 20706193






5 Functional Relationships(s)
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1 BACKGROUND: gp91(PHOX), a catalytic subunit of NAD(P)H oxidase, is involved in angiotensin II (Ang II)-induced superoxide (O2-) generation. Superoxides angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
2 BACKGROUND: gp91(PHOX), a catalytic subunit of NAD(P)H oxidase, is involved in angiotensin II (Ang II)-induced superoxide (O2-) generation. Superoxides angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
3 BACKGROUND: gp91(PHOX), a catalytic subunit of NAD(P)H oxidase, is involved in angiotensin II (Ang II)-induced superoxide (O2-) generation. Superoxides angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
4 BACKGROUND: gp91(PHOX), a catalytic subunit of NAD(P)H oxidase, is involved in angiotensin II (Ang II)-induced superoxide (O2-) generation. Superoxides angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus
5 CONCLUSION: These data suggest that an enhanced O2- activity and its interaction with NO contribute to the early developmental phase of Ang II-induced salt-sensitive hypertension.American Journal of Hypertension (2011). Superoxides angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus