Pub. Date : 2004
PMID : 20641916
3 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
1 | 4-[(18)F]Fluoropaclitaxel One of the mechanisms that tumor cells use to escape the cytotoxic effects of chemotherapeutic agents, such as Adriamycin, Vinca alkaloids, epipodophyllotoxins, actinomycin D, and paclitaxel (PAC), is to limit their presence inside the cells through the actions of P-glycoprotein (P-gp), a protein encoded by the multidrug resistance (MDR-1) gene (1, 2). | Podophyllotoxin | ATP binding cassette subfamily B member 1 | Homo sapiens |
2 | 4-[(18)F]Fluoropaclitaxel One of the mechanisms that tumor cells use to escape the cytotoxic effects of chemotherapeutic agents, such as Adriamycin, Vinca alkaloids, epipodophyllotoxins, actinomycin D, and paclitaxel (PAC), is to limit their presence inside the cells through the actions of P-glycoprotein (P-gp), a protein encoded by the multidrug resistance (MDR-1) gene (1, 2). | Podophyllotoxin | ATP binding cassette subfamily B member 1 | Homo sapiens |
3 | 4-[(18)F]Fluoropaclitaxel One of the mechanisms that tumor cells use to escape the cytotoxic effects of chemotherapeutic agents, such as Adriamycin, Vinca alkaloids, epipodophyllotoxins, actinomycin D, and paclitaxel (PAC), is to limit their presence inside the cells through the actions of P-glycoprotein (P-gp), a protein encoded by the multidrug resistance (MDR-1) gene (1, 2). | Podophyllotoxin | ATP binding cassette subfamily B member 1 | Homo sapiens |