Title : Opposing effects of cannabinoids and vanilloids on evoked quantal release at the frog neuromuscular junction.

Pub. Date : 2010 Apr 5

PMID : 20176082






4 Functional Relationships(s)
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1 Here we report that two cannabinoid type 1 receptor (CB1) agonists, WIN 55212-2 (WIN) and arachidonyl-2"-chloroethylamide (ACEA) have opposing effects on evoked quantal acetylcholine release - WIN decreased quantal content while ACEA increased quantal content. arachidonyl-2-chloroethylamide cannabinoid receptor 1 Homo sapiens
2 Here we report that two cannabinoid type 1 receptor (CB1) agonists, WIN 55212-2 (WIN) and arachidonyl-2"-chloroethylamide (ACEA) have opposing effects on evoked quantal acetylcholine release - WIN decreased quantal content while ACEA increased quantal content. arachidonyl-2-chloroethylamide cannabinoid receptor 1 Homo sapiens
3 Here we report that two cannabinoid type 1 receptor (CB1) agonists, WIN 55212-2 (WIN) and arachidonyl-2"-chloroethylamide (ACEA) have opposing effects on evoked quantal acetylcholine release - WIN decreased quantal content while ACEA increased quantal content. arachidonyl-2-chloroethylamide cannabinoid receptor 1 Homo sapiens
4 Here we report that two cannabinoid type 1 receptor (CB1) agonists, WIN 55212-2 (WIN) and arachidonyl-2"-chloroethylamide (ACEA) have opposing effects on evoked quantal acetylcholine release - WIN decreased quantal content while ACEA increased quantal content. arachidonyl-2-chloroethylamide cannabinoid receptor 1 Homo sapiens