Title : Modulation of K(ATP) currents in rat ventricular myocytes by hypoxia and a redox reaction.

Pub. Date : 2009 Oct

PMID : 19801996






2 Functional Relationships(s)
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1 Furthermore, in order to study the signaling pathways of the I(KATP) augmented by hypoxia and the redox agent, we applied a protein kinase C(PKC) inhibitor bisindolylmaleimide VI (BIM), a protein kinase G(PKG) inhibitor KT5823, a protein kinase A (PKA) inhibitor H-89, and Ca2+/calmodulin-dependent protein kinase II (CaMKII) inhibitors KN-62 and KN-93. bim protein kinase cAMP-activated catalytic subunit alpha Rattus norvegicus
2 Furthermore, in order to study the signaling pathways of the I(KATP) augmented by hypoxia and the redox agent, we applied a protein kinase C(PKC) inhibitor bisindolylmaleimide VI (BIM), a protein kinase G(PKG) inhibitor KT5823, a protein kinase A (PKA) inhibitor H-89, and Ca2+/calmodulin-dependent protein kinase II (CaMKII) inhibitors KN-62 and KN-93. bim protein kinase cAMP-activated catalytic subunit alpha Rattus norvegicus