Title : Bortezomib treatment of ovarian cancer cells mediates endoplasmic reticulum stress, cell cycle arrest, and apoptosis.

Pub. Date : 2009 Dec

PMID : 19039521






1 Functional Relationships(s)
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1 Bortezomib enhanced the sensitivity of ovarian cancer cells and tissue explants to an apoptosis-inducing TRAIL receptor antibody by upregulating the TRAIL receptor DR5. Bortezomib TNF receptor superfamily member 10b Homo sapiens